Mechanisms of HIV Drug Resistance Session Posters
Resistance to HIV-1 entry inhibitors
may occur by multiple molecular mechanisms
CJ Petropoulos
Mutations in HIV-1 RNase H domain
confer high-level resistance to nucleoside reverse transcriptase inhibitors
and provide novel insights into the mechanism of nucleotide excision-mediated
drug resistance
VK Pathak
Misincorporation of thymidine
analogues can increase drug susceptibility
M Götte
Effects of mutations associated
with suppression of zidovudine resistance in HIV-1 reverse transcriptase on
removal of tenofovir from blocked primer/template
J Lennerstrand
Kinetic mechanism of HIV-1 reverse
transcriptase catalyzed AZT excision
N Sluis-Cremer
Selective primer unblocking of
carbovir, lamivudine, tenofovir and zidovudine by wild-type HIV reverse transcriptase
with nucleotide and deoxynucleotide triphosphates
ER Lanier
The selective advantage of AZT-resistance
mutations can be demonstrated in vitro but is suppressed at high levels of PPi
AJ Smith
Bisphosphonate
inhibitors of nucleoside reverse transcriptase inhibitor excision
MA Parniak
Impaired rescue of chain-terminated
DNA synthesis associated with the L74V mutation in HIV-1 reverse transcriptase
MA Wainberg
Thermodynamic analysis of capravirine
binding to HIV reverse transcriptase and inhibition of DNA polymerase and RNase
H activities
K Klumpp
Novel mechanisms involved in non-nucleoside
reverse transcriptase inhibitor (NNRTI) resistance of both the subtype D HIV-1
isolate and reverse transcriptase derived from a drug-naive Ugandan
H Baird
Structural studies of benzophenone/HIV-1
RT complexes: insights into the potency of the next generation NNRTIs against
WT and mutant HIV-1
DK Stammers
Dinucleoside polyphosphates are
novel inhibitors of HIV-1 reverse transcriptase with increased potency against
enzymes containing AZT-resistance mutations
P Meyer
Natural resistance of HIV-2 to
zidovudine
JG García-Lerma
Potential reversion pathways of
Tyr 215 to Thr in HIV-1 reverse transcriptases having a dipeptide insertion
between codons 69 and 70: consequences for zidovudine and stavudine resistance
L Menéndez-Arias
The HIV-1 RT mutation H208Y combined
with T215Y causes hypersusceptibility to efavirenz
SA Clark
Effect of cell cycle arrest on
HIV susceptibility to RT inhibitors
F Clavel
Novel HIV drug resistance mechanism
leading to protease inhibitor (PI) resistance in response to a high genetic
barrier PI in vitro
M Nijhuis
Wide open 1.3 Å structure of the multidrug-resistant HIV-1
protease represents a novel drug target
LC Kovari
Predictors of selection of K65R:
tenofovir use and lack of TAMs
L Valer
In vitro antiviral interactions
among tenofovir, abacavir, lamivudine and didanosine
CL Tremblay
No evidence for stavudine resistance
due to nevirapine-selected mutation Y181C in HIV-1 reverse transcriptase in
a large
genotype/phenotype database
K Korn